Download Citation on ResearchGate | On Dec 1, , Edwin Arévalo Guerrero and others published Rol del strain miocárdico en. Abstract. RUIZ-MORI, Enrique; AYALA-BUSTAMANTE, Leonor; BURGOS- BUSTAMANTE, Jorge and PACHECO ROMAN, Cristian. Chemotherapy-induced . Resumen. RUIZ-MORI, Enrique; AYALA-BUSTAMANTE, Leonor; BURGOS- BUSTAMANTE, Jorge y PACHECO ROMAN, Cristian. Chemotherapy-induced.
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Old and current strategies for diagnosis and management. Role of biomarkers in chemotherapy-induced cardiotoxicity. Nat Rev Drug Discov. Lansigan F, Foss FM.
Tolerability of first-line therapy for metastatic renal quimioterqpia carcinoma. Acute vascular toxicity after combination chemotherapy with cisplatin, vinblastine, and bleomycin for testicular cancer. Venous thromboembolism and cancer: Cardiotoxicity of the anticancer therapeutic agent bortezomib.
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Cardiotoxicidad inducida por quimioterapia.
Chemotherapy is indicated in several phases of the antineoplastic treatment, either as neoadjuvant, adjuvant or palliative therapy. Congestive heart failure and left ventricular dysfunction complicating doxorubicin therapy.
Effect of arsenic trioxide on QT interval in patients with advanced malignancies. Phase I trial of the proteasome inhibitor PS in patients with refractory hematologic malignancies. Pharmacol Ther ; 2: Cardiotoxicity of chemotherapeutic agents: Troponins and natriuretic peptides in the monitoring of anthracycline cardiotoxicity.
Cardiotoxicidad por quimioterapia en el Instituto Nacional de Enfermedades Neoplásicas
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Letrozole therapy alone or in sequence with tamoxifen in women with breast cancer. Pentostatin increases the acute toxicity of high dose cyclophosphamide.
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Cardiotoxicidad inducida por quimioterapia
Periprocedural anticoagulation management of patients with venous thromboembolism. Apresentam atividade antitumoral significativa contra o carcinoma do colo uterino, linfomas e quimioterapis testiculares 4. Arterial pulse pressure variation predicting fluid responsiveness in critically ill patients. Cardiotoxicity is a known adverse event of oncologic therapy that many times limits a correct regimen of administration thus lowering efficacy; therefore, understanding pathophysiological and molecular bases is essential in order to determine preventive and therapeutical measures quimioterspia can allow a reduction in the incidence of toxicity, without interfering in the efficacy of the cancer therapy.
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